In a new study led by the Martinez-Sobrido Lab, researchers analyzed an H5N1 strain from a human infection and found nine mutations that make the virus replicate more efficiently in human cells and cause more severe disease in animal models, compared with a bovine strain. The team is now investigating which of these mutations drive increased virulence, while noting that FDA- approved antivirals remain effective, underscoring the lab's ongoing work on H5N1 pathogenesis and preparedness.
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